Referencing this question and its smart answer:
I'm still confused and the confusion has been in part fueled by "recent" Pearl/Hernán tweets (tweet, tweet and tweet).
Pearl's Ladder of causation and my rephrasing of the third W'question.
- Association - What is?
- Intervention - What if?
- Counterfactuals - What would be if?
Wouldn't interventions that are (perfect) randomized experiments, where two or more groups are treated, each group emulating counter-to-fact causes, not be squarely placed on rung 3.) - all along the Neyman-Rubin potential outcomes framework!? Isn't the potential outcomes framework and a framework for counterfactual outcomes conceptually identical? Pearl says:
Interventions change but do not contradict the observed world, because the world before and after the intervention entails time-distinct variables.
I can understand that interventions of the sort "If we take aspirin, will our headaches be cured" are rung 2.); but then they are not experiments, are they? (Perfect) randomized experiments are set up to compare a factual cause to a contrafactual one. What would be if I'd taken aspirin in comparison to not have taken any - or counterfactual: I've taken aspirin and others have been randomized to not to, so what is the difference in headache averages?
A part of the rung 2.) / rung 3.) "problem" can perhaps be traced back to different taxonomy. The "Harvard group" defines causal inference as:
Causal inference has a central role in public health; the determination that an association is causal indicates the possibility for intervention. (Glass, Goodman, Hernán, & Samet, 2013)
So in their eyes (Hernán's trichotomy: description, prediction (probably rung 1. on Pearl's ladder) and counterfactual prediction=(randomized, perfect) intervention=causal inference (Hernán, Hsu, & Healy, 2019)) they would most probably see interventions on rung 3.) (as "counterfactual interventions" perhaps?).
Which brings me to an interesting very recent intervention by Imbens (Imbens, 2019):
I would have liked to have seen a fourth rung of the ladder, dealing with “why,” or reverse causality questions.
Wouldn't that then call for a reformulation of the Ladder of causation?
- Association - What is?
- Intervention - What if?
- Counterfactual prediction/intervention (experimental or observational, if we could perfectly de-confound) - What would be if?
- Counterfactual explanation - Why?; by this I mean all that the other nice things we can do with counterfactuals (Chapter 8 -> The Book of Why; attribution, mediation, generalizability, etc., etc.)
Cheers